Our blood-brain-barrier is the capillary shield between our brain and circulating blood. It vitally restricts the transfer of all soluble proteins greater than 400 Da in the blood across to the brain. Our blood-brain barrier is remarkably similar to our intestinal barrier and under various circumstances, is compromised potentially with severe consequences.
This two-part webinar series with Dr Robert Silverman will reveal some alarming effects of a blood-brain barrier breach. He will also teach you how to prevent, recognise and treat a barrier failure. Within hours of concussion or other traumatic brain injuries, the blood-brain barrier and also the intestinal barrier fails. This allows the invasion of intestinal bacterial toxins, neuronal tissue-binding food proteins and cross-reactive food proteins into the once-protected nervous system.
Most bacterial toxins, some of which cause SIBO, contribute to systemic inflammation and cyclically keep the intestinal and blood-brain barriers open, even years after the exposure. Adding further damage, specific food proteins such as gliadin, milk butyrophilin and food aquaporins share structural similarity to brain structures such as our myelin, cerebellum, asialoganglioside, synapsin and the aquaporin.
Additionally, specific food lectins and agglutinins bind to myelin tissue. Patients with circulating food antibodies and/or lectins and agglutinins who experience blood-brain-barrier breaches are at risk of the reactive antibodies infiltrating their brain causing neurological damage. Once environmentally triggered antibodies cause brain tissue damage, then neurological autoantibodies can be created which further contribute to the neuroautoimmune process.
It is therefore critical to implement a pro-brain dietary protocol and treatment strategy to heal the broken blood-brain barrier. For the same reasons and at the same time as you would for intestinal leakage. By taking proactive strategies, the risk of neurological disease and degeneration occurring can be significantly reduced.
